It will be the BMO-MRW and it is understood to be the minimal distance involving the BMO and ILM within the ONH. In this video, anatomy of the ONH and GMPE is decoded from a neophyte user’s perspective, why BMO-MRW is much more essential than the traditional BMO-HRW for glaucoma assessment. This movie also highlights, just how utilizing the arrival of Anatomic Positioning System, scans were able to align general into the individual’s Fovea-to-BMO-center (FoBMOC) axis at every follow-up, for precisely detecting changes, no more than 1 micron in BMO-MRW, hence producing a unique world in diagnosing glaucoma and detecting glaucomatous development with accuracy. Marin-Amat syndrome is an unusual acquired oculofacial synkinesis initially reported in 1918. It exhibits as involuntary eyelid closure on jaw orifice or on lateral movement of this jaw following a peripheral facial nerve medical photography palsy. The increased orbicularis tone as a result of aberrant connections between the cranial neurological (CN) V and CN VII leads to an unhealthy wink with major psychosocial impact. Many cases in literature were often observed or administered botulinum toxin injection towards the orbicularis muscle mass. There are few sporadic reports of surgical interventions with effective results.Hence there is a necessity to create understanding regarding different settings of handling of this rare entity. We provide a video clip in the clinical presentation and management of six such patients, of who one ended up being bilateral. Five patients were females. Terrible facial neurological paralysis and Bell’s palsy was once identified in one single and five clients respectively. The mean age was 52 ± 9.48 years. The mean MRD (margin reflex length) 1 and MRD 2 was 3.17 ± 0.60 and 5.33± 0.65 mm correspondingly. On smiling or on movement regarding the jaw the MRD 1 and 2 was reduced by 2. 50±0.40 and 1.50+/-0.40 mm respectively. Regarding the six patients four patients opted for nil intervention. Botulinum toxin injection and preseptal orbicularis resection when you look at the upper and lower eyelid along side blepharoplasty was performed in 1 client each. Satisfactory lowering of the synkinetic motion had been achieved both in presymptomatic infectors . Marin-Amat syndrome is a rare usually underdiagnosed synkinetic disorder following peripheral facial nerve palsy. Botulinum toxin shot and preseptal orbicularis resection are viable administration options. The movie demonstrates the tips to ascertain the anatomical integrity of this world and ideas to avoid suture bites through the choroid in a corneo-scleral tear repair. Identification of important landmarks helps in the establishment of anatomical integrity. Therefore, the limbal area of tear is very first sutured with 10-O nylon. The extent of injury on the sclera is inspected on the other end of the tear. Second limbal suture during the opposite end associated with the tear is taken, followed closely by dividing the corneal degree of tear by guideline of one half and segmental suturing with 10-O nylon. Then conjunctival peritomy is done to explore the scleral extent and the uveal muscle prolapse. Blunt and atraumatic straight back tip of Weckel sponge is used perpendicular towards the plane associated with the sclera to push the choroid straight back aiding the scleral bite. Sclera is sutured with 9-O nylon suture using attention to not ever range from the choroidal structure. Air shot is performed to check for any injury leak. Side port is hydrated, and corneal sutures are buried. The conjunctiva is secured with fibrin glue. Anterior chamber is formed with atmosphere bubble. Povidone iodine is instilled and BCL placed. 1. Suturing the landmark places first; 2. Exploring the level of wound; 3. Segmental suturing of the cornea; 4. pressing the choroid back to avoid bites through it while suturing sclera; 5. Air shot to check for wound leaks; 6. Anterior chamber formation with air at the end. Open up TAS-120 concentration globe injury is a significant sight threatening problem. Full-thickness, non-selfsealing corneal lacerations require fix into the operating room. During fix, debridement of this wound is an important action. Incarceration for the intraocular structures when you look at the wound eg. Iris, lens pill, vitreous leads to improper healing if not removed precisely. To show the manner of wound debridement in open world injury. Manual removal of incarcerated tissue articles causes partial cleaning & enormous grip on intra ocular contents. In this video clip, we have tried vitrectomy cutter with greater vacuum cleaner for cleaning the sides of this wound especially in the posterior aspect and debri removal, followed closely by effortless suturing. All muscle when you look at the wound edges are removed effectively without any grip on intraocular contents. Acute corneal hydrops is a sight threatening complication of corneal ectasia like keratoconus, keratoconus, keratoglobus, Pellucid limited degeneration, Terrien’s limited degeneration and post refractive surgery keratectasia. The connected risk facets for development of corneal hydrops (CH) are early start of keratoconus, microtrauma involving contact lens use, eye rubbing, allergic conjunctivitis, atopy, and Down’s problem. Aided by the conservative method of handling of CH, it takes longer time (in months) for corneal oedema getting resolved and there is improvement vascularization and scar tissue formation. This movie presents the easy means of using compression sutures along with pneumodescemetopexy by intracameral environment shot for handling of CH. It resulted in rapid quality of corneal oedema. It really is a straightforward strategy, with no need of unique gases like C3F8 or SF6 and can be easily performed at an extremely basic put up.